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Figure 4-4 A Schematic diagram of the main neurotransmitter pathways and their effects in the cortical basal ganglia thalamic circuits The blue lines indicate neurons with excitatory effects, while the black lines indicate inhibitory in uences The internal (medial) segment of the globus pallidus (MGP) and the zona reticulata of the substantia nigra (SNpr) are believed to act as one entity that projects via GABA-containing neurons to the thalamus (ventrolateral and ventroanterior nuclei) and to the pedunculopontine nuclei (not shown) Dopaminergic neurons (DA) arising in the pars compacta of the substantia nigra (SNpc) have an excitatory in uence on the direct striatopallidal bers (via D1 receptors) and an inhibitory effect on the indirect striatopallidal bers (via D2 receptors) that project to the external (lateral) pallidum (LGP) and subthalamic nucleus (STN) Dotted lines in the subsequent gures denote a reduction in activity of the pathway (See text) B Corresponding physiologic state as conceptulazied in Parkinson disease, in which hypokinesia is the main nding as a result of reduced dopamine input from the substantia nigra and pars compacta to the striatum via the direct pathway, which results in withdrawal of inhibitory activity of the globus pallidus and, in turn, increased inhibitory drive on the thalamic nuclei, which reduces input to the cortical motor system C Schematic diagram of the theorized mechanism in Huntington disease, a hyperkinetic movement disorder resulting from reduced inhibition by the striatum within the indirect pathway, overdriving of the subthalamic nulceus, and causing excess activity in thalamocortical circuits (See text)

During the past three decades, a series of pharmacologic observations some mentioned above have considerably enhanced our understanding of basal ganglionic function and led to a rational treatment of Parkinson disease and other extrapyramidal syndromes Whereas physiologists had for years failed to discover the functions of the basal ganglia by stimulation and crude ablation experiments, clinicians became aware that the use of certain drugs, such as reserpine and the phenothiazines, regularly produced extrapyramidal syndromes (eg, parkinsonism, choreoathetosis, dystonia) These observations greatly stimulated the study of central nervous system (CNS) transmitter substances in general The current view is that the integrated basal ganglionic control of movement can be best understood by considering, in the context of the anatomy described above, the physiologic effects of neurotransmitters that convey the signals between cortex, striatum, globus pallidus, subthalamic nucleus, substantia nigra, and thalamus The most important neurotransmitter substances from the point of view of basal ganglionic function are glutamate, GABA, dopamine, acetylcholine, and serotonin Their roles are sum-

Analyzer and BEx Analyzer you should be on at least SPS12 On a lower SPS than this, you will be able to create both local and global variants on the BEx Analyzer but only the local variant on the BEx Web Analyzer So to recap, there are three different data stores available in NW BI 70: A SAP BW 3x Runtime = VARI B SAP BW 3x Runtime within NW BI 70 = RSRVARIANT C NW BI 70 Runtime = RSRPARAMETRIZA In order to migrate variants from tables for A to tables for B you can utilize the ABAP program RSR_VARIANT_XPRA In order to migrate query variants from data store B to data store C or vice versa you will utilize another migration program You need to initially import the transport available that has this specific migration component included Make sure you review the process within your system so that you do not write over existing programs within your system Once you are done importing the SBC transport with the programs installed, you then go to transaction code SE38 or SA38 and run the program Z_MIGRATE_VARIANTS Once you have executed this program, you see the execution screen shown in the following illustration

marized in Fig 4-4 A more complete account of this subject than is possible here may be found in the reviews of Penney and Young, Alexander and Crutcher, and of Rao referenced at the end of this chapter The following is what is known with a fair degree of certainty Glutamate is the neurotransmitter of the excitatory projections from the cortex to the striatum and of the excitatory neurons of the subthalamic nucleus GABA is the inhibitory neurotransmitter of striatal, pallidal, and substantia nigra (pars reticulata) projection neurons Acetylcholine (ACh), long established as the neurotransmitter at the neuromuscular junction and the autonomic ganglia, is also physiologically active in the basal ganglia The highest concentration of ACh as well as of the enzymes necessary for its synthesis and degradation (choline acetyl transferase and acetylcholinesterase) is in the striatum Acetylcholine is synthesized and released by the large but sparse (Golgi type 2) nonspiny striatal neurons It has a mixed but mainly excitatory effect on the more numerous spiny neurons within the putamen that constitute the main origin of the direct and indirect pathways described above Dopamine, by contrast, has both inhibitory and excitatory effects on these neurons, as detailed below It is likely that the effectiveness of atropinic

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